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You are here: Home >Extracts> Herbal & Plant Extracts > Dihydromyricetin 98%
wholesale Dihydromyricetin 98%
FOB Price:USD434.48/kg
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Appearance: White crystalline powder
Sources: Ampelopsis grossedentata
CAS Number: 27200-12-0
MOQ: 1kg
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-- -- USD434.48  /kg
1.00kg 24.00kg USD517.24  /kg
25.00kg 100.00kg USD434.48  /kg
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Appearance White crystalline powder
Sources Ampelopsis grossedentata
CAS Number 27200-12-0
MOQ 1kg
Specification 98%
Molecular Formula C15H12 O8
Molecular Weight 320  
E.Coli Negative
Salmonella Negative
Bioactive Ingredients Dihydromyricetin
Mesh NLT 98% through 80 mesh
Dihydromyricetin 98% Detail
Dihydromyricetin DMY
Latin Name: Myrica rubra Zucc; Ampolopsis grossedentata W.T.Wang.
Specification: Dihydromyricetin DMY 98% (HPLC)
Molecular weight: 320
Molecular formula: C15H12 O8
CAS No: 27200-12--0
Product type: white powdered extract
Part of the plant used: Leaves , bark
Extract method: Ethanol / water

Vine tea is one of a rare plant. Botanical name is ampelopsis grossedentata, it is one kind of typical tea plant, "Chinese Herbal Medicine Collection" wrote that the vine tea (Teng Cha )has a glycol flavor, the nature is cool; it can alleviate fevers, dis-intoxicate, cure rheumatism, strengthen physique, diuresize, and many other functions. The vine tea (Teng Cha) contains rich flvones, amino acids, vitamins; proteins and essential trace element, in which the content of total flavones, and Dihydromyricetin elementis are especially high.

Packing Detail:
Packed in paper-drums and two plastic-bags inside. Net weight:25kgs/drum.

Storage situation:

Stored in a cool and dry well-closed container, keep away from moisture and strong light/heat.

UCLA researchers have identified how a component of an ancient Chinese herbal anti-hangover medicine called dihydromyricetin, isolated from the plant Hovenia, counteracts acute alcohol intoxication 

and withdrawal symptoms.

The research team found that dihydromyricetin blocks the action of alcohol on the brain and neurons and also reduces

 voluntary alcohol consumption, with no major side effects, in an early study with rats. Specifically, dihydromyricetin

 inhibited alcohol's effect on the brain's GABAA receptors, specific sites targeted by chemicals from brain cells. 

Alcohol normally enhances the GABAA receptors' influence in slowing brain cell activity, reducing the ability to 

communicate and increasing sleepiness - common symptoms of drunkenness.

Dihydromyricetin (DHM; 1 mg/kg, i.p. injection), a flavonoid component of herbal medicines, counteracted acute 

alcohol (EtOH) intoxication, and also withdrawal signs in rats including tolerance, increased anxiety, and seizure 

susceptibility; DHM greatly reduced EtOH consumption in an intermittent voluntary EtOH intake paradigm in rats. 

GABAA receptors (GABAARs) are major targets of acute and chronic EtOH actions on the brain. At the cellular levels, 

DHM (1 μm) antagonized both acute EtOH-induced potentiation of GABAARs and EtOH exposure/withdrawal-induced GABAAR plasticity, including alterations in responsiveness of extrasynaptic and postsynaptic GABAARs to acute EtOH and, most importantly, increases in GABAAR α4 subunit expression in hippocampus and cultured neurons.

The research team determined that dihydromyricetin may provide a molecular target and cellular mechanism to 

counteract alcohol intoxication and dependence, leading to new therapeutic treatments - all based on an ancient 

"folk medicine" treatment that has been used by humans for at least 500 years. 

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